![]() I have tried to find out how VST’s like PianoTeq handle this from different controllers. Maybe it is a complicated task due to i) as you say it does not really excist a standard for midi-implementation of triple sensor action or ii) the NS3C also have an organ and a synth engine which may be affected by the triple sensor midi-stream. After testing they concluded they did not handle it but would not promise to if or when they would work on an adaptation. They wanted me to send a midi file from the Kawai to verify because they thought the NS3C would handle it correctly. I had discussions with Nord about this a couple of years ago based upon my test with a Kawai MP11. This would only work from a NP 4/5 or Grand. And seem to interpret them in a way that sounds like fast repetition. While you are right for other brands, Nord triple sensor keyboards seem to send Note-On, Note-Off, Note-On, Note-Off even from a triple sensor keyboard. We heard more than once that Nord handles its triple sensor MIDI commands differently than e.g. (But in any case, I am not a classical musician with a significant need to quickly retrigger a single note repeatedly, so the triple sensor wouldn't be too much of a benefit in my case - unless there is something else that the triple sensor provides that I am unaware of.) JVSalazar Posts: 29 Joined:, 21:09 Country: Has thanked: 1 time Been thanked: 2 timesįZiegler The thing with the triple sensor keyboard doesn't seem to be true overall. Why would this be the case? Wouldn't a note being activated by the triple sensor just translate to a regular MIDI note that could be processed universally? Or is triple sensor MIDI data inherently different from notes generated from a non-triple sensor? If you combine a NS3C with a NP5 you will have the triple sensor benefit when playing the the pianos of the NP5 directly. The Nord Stage 3 Compact will not benefit of this when controlled by a triple sensor keyboard. Valpurgis wrote:There is one point that nobody other than me seem to bother about, and I find it strange: Triple sensor keybeds make it easier to restrike the sound without lifting the key above the upper sensor. JVSalazar Posts: 29 Joined:, 21:09 Country: Has thanked: 1 time Been thanked: 2 times I don't enjoy playing TP100 keybeds though, so most boards with that keybed are out of the question for me. Maybe there's another option for a relatively portable weighted keyboard that I could use to trigger Nord sounds. But when using both together I don't know if the NP5 would be anything more than purely a MIDI controller. ![]() ![]() Although now I suppose the NP5 could be used as a backup if my NS3C fails on stage, or I could use it in cases where I only need a single board for gigs and would prefer a weighted action over the NS3C functionality. Is there anything that the NP5 can do that the NS3C can't, functionality wise? The only use I can foresee is that I can add more XL pianos on the NP5 that don't fit on my NS3C, but given the price point, I don't know if just that benefit would be worth it. My question now is that if a NP5 worth it, given that I already have a NS3C. As much as I like a few of the sounds on there that the Nord doesn't have, I'm probably going to get rid of because of this. I already have a CP73, but to be blunt, the velocity curve of the keybed just sucks and has very little dynamic range no matter which touch setting I use, especially when using the MIDI out to trigger the Nord pianos. The reason why I'm looking at the NP5 73 is that I want a weighted action to play piano parts, and so far it is the most portable keyboard I've found with an action that I enjoy. I currently have a NS3C and am considering getting a Nord Piano 5 73 for a relatively portable dual board setup.
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One of the probable reasons for drug resistance is biofilm production as biofilm is made up of multi-layered cell clusters embedded in a matrix of extracellular polysaccharide (EPS), which provide adherence to the bacteria. aureus (MRSA), which consequently presents difficult therapeutic problems and biofilm-producing strains make it worse. aureus rapidly develops resistance to many antimicrobial agents, and the classic example is methicillin resistance S. aureus often hemolyze blood, coagulate plasma, and produce a variety of extracellular enzymes, toxins, and biofilm. aureus is one of the most common causes of hospital- and community-acquired infections. aureus is a member of the normal microbiota of the skin and mucous membranes of humans, therefore, cause suppuration, abscess formation, a variety of pyogenic infections, and even fatal septicemia. aureus is Gram-positive spherical cocci, usually arranged in grapelike irregular. 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